CMT1I

POLR3B | 2021

Illustration of a DNA double helix overlaid on nerve cells, with text reading ‘Experts in CMT: CMT Genes and Subtypes Database

CMT1I is caused by mutations in the POLR3B gene. This gene encodes (provides instructions for)a subunit of RNA polymerase III, which plays a crucial role in regulating the expression of genes essential for nervous system development and function.

CMT1I is autosomal dominant, meaning that just one of the gene’s two copies needs a mutation to cause this subtype.

Clinical Features

Symptoms typically begin in early childhood. In many individuals, symptoms first affect the lower extremities and later involve the upper limbs. Some individuals also show features of central nervous system involvement. Nerve conduction studies show slowed conduction velocities consistent with a demyelinating CMT.

CMT1I symptoms may include:

  • Symptoms typically begin in early childhood
  • First symptoms typically appear in the lower extremities
  • Delayed motor development or delayed walking
  • Weakness in the feet and lower legs
  • Muscle atrophy
  • Foot drop
  • A steppage-style or unsteady walking pattern
  • Reduced sensation
  • Reduced or absent reflexes
  • Foot deformities, including high arches and hammertoes (clawed toes)
  • Clawing of the fingers
  • Balance and coordination difficulties, including ataxia
  • Speech difficulties, including dysarthria or delayed speech
  • Tremor or abnormal movements
  • Additional symptoms not listed here

Disease Course

CMT1I shows variability in severity and progression.  Some individuals experience a relatively mild course, while others develop a more severe presentation. Progression is generally slow, and life expectancy is not reduced.

Clinical Basics

Subtype
CMT1I

Classification
CMT1

Neuropathy Type
Demyelinating

Inheritance Pattern
Autosomal Dominant

Genetic Context

HGNC-Approved Gene Symbol
POLR3B

Gene Full Name
RNA Polymerase III Subunit B

HGNC Gene Alias(es)
RPC2, FLJ10388, C128

Chromosome
12q23.3

Zygosity of Responsible Variant
Heterozygous

Mitochondrial Involvement
No

ClinVar Pathogenic Variants

View CMT1I ClinVar Variants

More Info

Research Opportunity

CMT Natural History Study

Original Discovery Publication

Publication Title

De novo Variants in POLR3B Cause Ataxia, Spasticity, and Demyelinating Neuropathy

Authors

Oegema, R., van Binsbergen, E., Baskin, B., Bernard, G., Fribourg, S., Coulombe, B., & Yoon, G

Publication Date
January 7, 2021

Updated: February 1, 2026 | By: K. Raymond

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