CMTX3

ins(X;8)(q27.1;q24.3) | 2016

What Is CMTX3?

CMTX3 is a type of CMT caused by an insertion, a form of chromosome rearrangement in which a segment of genetic material ends up somewhere it does not belong. In CMTX3, a 78 kb segment normally found on chromosome 8 (8q24.3) has been inserted into the X chromosome (at Xq27.1). The material moved in one direction only, with nothing exchanged in return, which is what separates an insertion from a reciprocal translocation.

CMTX3 is X-linked recessive. This means the change responsible sits on the X chromosome, and in people with two X chromosomes (chromosomal females), both copies must carry it to cause CMTX3. For individuals with one X and one Y chromosome (chromosomal males), a single affected copy is sufficient. CMTX3 differs from most X-linked forms of CMT. Its cause is not a mutation inside a gene. The segment from chromosome 8 was inserted into a stretch of the X chromosome that lies between genes.

Reading the Notation

Every other CMT subtype with a known cause is related to a mutation in a single gene. The gene is identified by its HUGO Gene Nomenclature Committee (HGNC) gene symbol. But CMTX3 is the exception. HGNC assigns symbols to genes, not chromosomal rearrangements. So, CMTX3 uses the International System for Human Cytogenomic Nomenclature (ISCN) notation instead. The ISCN standard covers changes at the chromosome level rather than at the gene level.

Read it left to right. ins gives the type of change, an insertion. (X;8) names the chromosomes involved, the one receiving the material first, and the one donating it second. (q27.1;q24.3) gives the band on each, in that same order. Together, they say that genetic material from band q24.3 of chromosome 8 has been inserted into band q27.1 of the X chromosome.

The notation as published is longer: der(X)dir ins(X;8)(q27.1;q24.3). The der(X) means that the X chromosome is the altered one, and dir means that the inserted segment kept its original orientation rather than being flipped end to end.

Because ISCN names the receiving chromosome first, the notation opens with X while a plain-language description of the same event opens with chromosome 8. Both describe the same insertion.

Clinical Features

CMTX3 symptoms begin by the early teens. They typically begin in the lower extremities and progress over time to involve the upper limbs. Nerve conduction studies usually show slowed conduction velocities, consistent with a demyelinating form of CMT.

CMTX3 symptoms may include:

  • Weakness in the feet and lower legs
  • Muscle atrophy
  • Foot drop
  • A steppage-style walking pattern
  • Reduced sensation
  • Reduced or absent reflexes
  • Foot deformities, including high arches and hammertoes (clawed toes)
  • Progressive involvement of the hands and forearms
  • Difficulty with fine motor skills and manual dexterity
  • Balance difficulties
  • Additional symptoms not listed here

Disease Course

CMTX3 shows wide variability in severity and progression. Some individuals are mildly affected, while others develop a more severe disease. Onset is by the early teens, but disease progression is generally slow, and life expectancy is not reduced.

Clinical Basics

Subtype
CMTX3

Classification
CMTX

Neuropathy Type
Demyelinating

Inheritance Pattern
X-linked recessive

Genetic Context

ISCN Notation
ins(X;8)(q27.1;q24.3)

Rearrangement Description
78 kb Interchromosomal Insertion at Xq27.1

Chromosome
Xq27.1

Zygosity of Responsible Variant
Hemizygous (Male) / Homozygous (Female)

Mitochondrial Involvement
No

Variant Mechanism

Unknown

Details

Mechanistic basis:
Gene unknown

Confidence:
Low

Prediction:
The literature does not attribute CMTX3 to a coding mutation. It arises from a large interchromosomal insertion of chromosome 8q24.3 sequence into Xq27.1 (der(X)dir ins(X;8)(q27.1;q24.3)) (Brewer et al.), a non-coding structural variant in a gene desert. The predicted mechanism is a positional or regulatory effect on a nearby gene, but no causative single-gene or defined loss- or gain-of-function mechanism has been established, so the mechanism remains unresolved.

Rationale:
CMTX3 arises from a non-coding Xq27.1/8q24.3 insertion (der(X)dir ins(X;8)(q27.1;q24.3)) thought to exert a position or regulatory effect, but with no causative single-gene identified, the mechanism remains unresolved at low confidence.

CMTX3 OMIM Entry

CMTX3 OMIM

More Info

CMTX3 Research Opportunity

CMT Natural History Study

Original Discovery Publication

Publication Title

Whole Genome Sequencing Identifies a 78 kb Insertion from Chromosome 8 as the Cause of Charcot-Marie-Tooth Neuropathy CMTX3

Authors

Brewer, M. H., Chaudhry, R., Qi, J., Kidambi, A., Drew, A. P., Menezes, M. P., Ryan, M. M., Farrar, M. A., Mowat, D., Subramanian, G. M., Young, H. K., Züchner, S., Reddel, S. W., Nicholson, G. A., & Kennerson, M. L.

Publication Date
July 20, 2016

Updated: August 3, 2026 | By: K. Raymond

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